Motion sickness habituation represents a core neuroplastic model for investigating vestibular disorder adaptation, yet its brain-region-specific temporal molecular mechanisms remain poorly defined. Here, we adopted multi-brain-region proteomic screening combined with time-series molecular validation to characterize key molecular dynamics during motion sickness habituation. Adult mice received 15 consecutive days of sinusoidal rotational stimulation, and model establishment was verified via four...
Source: PubMed - functional neurological disorder
·Published 23 Oct 2026 ·Imported 23 Oct 2026
Relevancy: Not reviewed
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